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cvs 11  (ATCC)


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    Structured Review

    ATCC cvs 11
    Cvs 11, supplied by ATCC, used in various techniques. Bioz Stars score: 94/100, based on 67 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/cvs 11/product/ATCC
    Average 94 stars, based on 67 article reviews
    cvs 11 - by Bioz Stars, 2026-04
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    ATCC rabies virus cvs 11
    The characterization of LINC2781. (A) A schematic representation of the human LINC2781 transcribed from the XLOC_262866 gene; (B) SH-SY5Y and THP-1 cells were infected with CVB5 at increasing MOIs for 24 h or CVB5 (MOI = 1) for the indicated time intervals. The expression of LINC2781 was measured by RT-qPCR; (C) SH-SY5Y cells were transfected with increasing amounts of CVB5 RNA or plasmids (2 µg) encoding viral nonstructural proteins. The expression of LINC2781 was measured by RT-qPCR; (D) SH-SY5Y cells were infected with CVB5, CVA16, EV71, HSV-1, and <t>RABV</t> (MOI = 1) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (E) SH-SY5Y cells were stimulated with different amounts of poly (I:C) or poly (dG:dC) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (F) Various cell lines were infected with CVB5 (MOI = 1) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (G) Three-day-old BALB/c mice were infected with CVB5 (20 LD50) for 10 days. The expression of LINC2781 was measured by RT-qPCR. (H) SH-SY5Y cells were treated with increasing amounts of IFN-β, IFN-γ, LPS, and TNF-α for 24 h. The expression of LINC2781 was measured by RT-qPCR. Biologically independent experiments ( n = 3) were conducted, and all data were shown as mean ± SD. Student’s t -test was used to detect significant differences, with P ≤ 0.05 (*), P ≤ 0.01 (**), P ≤ 0.001 (***), and ns for no significant difference.
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    ATCC rabv cvs 11 strain
    The characterization of LINC2781. (A) A schematic representation of the human LINC2781 transcribed from the XLOC_262866 gene; (B) SH-SY5Y and THP-1 cells were infected with CVB5 at increasing MOIs for 24 h or CVB5 (MOI = 1) for the indicated time intervals. The expression of LINC2781 was measured by RT-qPCR; (C) SH-SY5Y cells were transfected with increasing amounts of CVB5 RNA or plasmids (2 µg) encoding viral nonstructural proteins. The expression of LINC2781 was measured by RT-qPCR; (D) SH-SY5Y cells were infected with CVB5, CVA16, EV71, HSV-1, and <t>RABV</t> (MOI = 1) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (E) SH-SY5Y cells were stimulated with different amounts of poly (I:C) or poly (dG:dC) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (F) Various cell lines were infected with CVB5 (MOI = 1) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (G) Three-day-old BALB/c mice were infected with CVB5 (20 LD50) for 10 days. The expression of LINC2781 was measured by RT-qPCR. (H) SH-SY5Y cells were treated with increasing amounts of IFN-β, IFN-γ, LPS, and TNF-α for 24 h. The expression of LINC2781 was measured by RT-qPCR. Biologically independent experiments ( n = 3) were conducted, and all data were shown as mean ± SD. Student’s t -test was used to detect significant differences, with P ≤ 0.05 (*), P ≤ 0.01 (**), P ≤ 0.001 (***), and ns for no significant difference.
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    ANSES laboratories strain cvs-11
    The characterization of LINC2781. (A) A schematic representation of the human LINC2781 transcribed from the XLOC_262866 gene; (B) SH-SY5Y and THP-1 cells were infected with CVB5 at increasing MOIs for 24 h or CVB5 (MOI = 1) for the indicated time intervals. The expression of LINC2781 was measured by RT-qPCR; (C) SH-SY5Y cells were transfected with increasing amounts of CVB5 RNA or plasmids (2 µg) encoding viral nonstructural proteins. The expression of LINC2781 was measured by RT-qPCR; (D) SH-SY5Y cells were infected with CVB5, CVA16, EV71, HSV-1, and <t>RABV</t> (MOI = 1) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (E) SH-SY5Y cells were stimulated with different amounts of poly (I:C) or poly (dG:dC) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (F) Various cell lines were infected with CVB5 (MOI = 1) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (G) Three-day-old BALB/c mice were infected with CVB5 (20 LD50) for 10 days. The expression of LINC2781 was measured by RT-qPCR. (H) SH-SY5Y cells were treated with increasing amounts of IFN-β, IFN-γ, LPS, and TNF-α for 24 h. The expression of LINC2781 was measured by RT-qPCR. Biologically independent experiments ( n = 3) were conducted, and all data were shown as mean ± SD. Student’s t -test was used to detect significant differences, with P ≤ 0.05 (*), P ≤ 0.01 (**), P ≤ 0.001 (***), and ns for no significant difference.
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    Changchun BCHT Biotechnology Co Ltd rabv strain cvs-11
    The characterization of LINC2781. (A) A schematic representation of the human LINC2781 transcribed from the XLOC_262866 gene; (B) SH-SY5Y and THP-1 cells were infected with CVB5 at increasing MOIs for 24 h or CVB5 (MOI = 1) for the indicated time intervals. The expression of LINC2781 was measured by RT-qPCR; (C) SH-SY5Y cells were transfected with increasing amounts of CVB5 RNA or plasmids (2 µg) encoding viral nonstructural proteins. The expression of LINC2781 was measured by RT-qPCR; (D) SH-SY5Y cells were infected with CVB5, CVA16, EV71, HSV-1, and <t>RABV</t> (MOI = 1) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (E) SH-SY5Y cells were stimulated with different amounts of poly (I:C) or poly (dG:dC) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (F) Various cell lines were infected with CVB5 (MOI = 1) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (G) Three-day-old BALB/c mice were infected with CVB5 (20 LD50) for 10 days. The expression of LINC2781 was measured by RT-qPCR. (H) SH-SY5Y cells were treated with increasing amounts of IFN-β, IFN-γ, LPS, and TNF-α for 24 h. The expression of LINC2781 was measured by RT-qPCR. Biologically independent experiments ( n = 3) were conducted, and all data were shown as mean ± SD. Student’s t -test was used to detect significant differences, with P ≤ 0.05 (*), P ≤ 0.01 (**), P ≤ 0.001 (***), and ns for no significant difference.
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    The characterization of LINC2781. (A) A schematic representation of the human LINC2781 transcribed from the XLOC_262866 gene; (B) SH-SY5Y and THP-1 cells were infected with CVB5 at increasing MOIs for 24 h or CVB5 (MOI = 1) for the indicated time intervals. The expression of LINC2781 was measured by RT-qPCR; (C) SH-SY5Y cells were transfected with increasing amounts of CVB5 RNA or plasmids (2 µg) encoding viral nonstructural proteins. The expression of LINC2781 was measured by RT-qPCR; (D) SH-SY5Y cells were infected with CVB5, CVA16, EV71, HSV-1, and RABV (MOI = 1) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (E) SH-SY5Y cells were stimulated with different amounts of poly (I:C) or poly (dG:dC) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (F) Various cell lines were infected with CVB5 (MOI = 1) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (G) Three-day-old BALB/c mice were infected with CVB5 (20 LD50) for 10 days. The expression of LINC2781 was measured by RT-qPCR. (H) SH-SY5Y cells were treated with increasing amounts of IFN-β, IFN-γ, LPS, and TNF-α for 24 h. The expression of LINC2781 was measured by RT-qPCR. Biologically independent experiments ( n = 3) were conducted, and all data were shown as mean ± SD. Student’s t -test was used to detect significant differences, with P ≤ 0.05 (*), P ≤ 0.01 (**), P ≤ 0.001 (***), and ns for no significant difference.

    Journal: mSphere

    Article Title: LINC2781 enhances antiviral immunity against coxsackievirus B5 infection by activating the JAK-STAT pathway and blocking G3BP2-mediated STAT1 degradation

    doi: 10.1128/msphere.00062-25

    Figure Lengend Snippet: The characterization of LINC2781. (A) A schematic representation of the human LINC2781 transcribed from the XLOC_262866 gene; (B) SH-SY5Y and THP-1 cells were infected with CVB5 at increasing MOIs for 24 h or CVB5 (MOI = 1) for the indicated time intervals. The expression of LINC2781 was measured by RT-qPCR; (C) SH-SY5Y cells were transfected with increasing amounts of CVB5 RNA or plasmids (2 µg) encoding viral nonstructural proteins. The expression of LINC2781 was measured by RT-qPCR; (D) SH-SY5Y cells were infected with CVB5, CVA16, EV71, HSV-1, and RABV (MOI = 1) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (E) SH-SY5Y cells were stimulated with different amounts of poly (I:C) or poly (dG:dC) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (F) Various cell lines were infected with CVB5 (MOI = 1) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (G) Three-day-old BALB/c mice were infected with CVB5 (20 LD50) for 10 days. The expression of LINC2781 was measured by RT-qPCR. (H) SH-SY5Y cells were treated with increasing amounts of IFN-β, IFN-γ, LPS, and TNF-α for 24 h. The expression of LINC2781 was measured by RT-qPCR. Biologically independent experiments ( n = 3) were conducted, and all data were shown as mean ± SD. Student’s t -test was used to detect significant differences, with P ≤ 0.05 (*), P ≤ 0.01 (**), P ≤ 0.001 (***), and ns for no significant difference.

    Article Snippet: Coxsackievirus A16 (CVA16) strain 74/YN/2016 (GenBank: KY440934.1 ), enterovirus A71 (EV71) strain RA330/YN/CHN/2009 (GenBank: MK028135.1 ), human herpes virus 1 (HSV-1) isolate ZW6 (GenBank: KX424525.1 KX424525.1 ), and rabies virus CVS-11 (RABV, ATCC VR 959) were stored in our laboratory.

    Techniques: Infection, Expressing, Quantitative RT-PCR, Transfection